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In the current paper, realizing that MYC, a transcription element that binds the genome through sites that are identical to the binding sites of BMAL1, the team hypothesized that aberrant MYC expression perturbs the clock by deregulating the different parts of the circadian network in malignancy cells. They compared gene expression from tumors of 740 sufferers to overall patient survival to reach at these correlations. Furthermore, re-expression of BMAL1 in neuroblastoma cell lines suppresses their ability to replicate. MYC also profoundly alters the oscillations of glucose fat burning capacity and perturbs the intake of glutamine, both important metabolic molecules for cells. ‘We thought that some tumor cells' metabolites oscillate plus some don't, but we’d not tested it,’ Altman recalled.